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Author: m miral dizdar

Displaying records 31 to 40 of 63 records.
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31. New functions of XPC in the protection of human skin cells from oxidative damage
Published: 9/7/2006
Authors: M. D'Errico, E. Parlanti, M. Teson, B. M. Bernardes de Jesus, P. Degan, A. Calcagnile, Pawel Jaruga, M. Bjoras, M. Crescenzi, A. M. Pedrini, J.-M. Egly, G. Zambruno, M. Stefanini, M Miral Dizdar, E. Dogliotti
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=907981

32. Oxidative changes in the DNA of stroma and epithelium from the female breast: potential implications for breast cancer
Published: 8/1/2006
Authors: D. C. Malins, K. M. Anderson, Pawel Jaruga, C. R. Ramsey, N. K. Gilman, W. M. Green, S. W. Rostad, J. T. Emerman, M Miral Dizdar
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=908170

33. Biomarkers Signal Contaminant Effects on the Organs of English Sole (Parophrys vetulus) from Puget Sound
Published: 6/1/2006
Authors: D. C. Malins, K. M. Anderson, J. J. Stegeman, Pawel Jaruga, V. M. Green, N. K. Gilman, M Miral Dizdar
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=908169

34. Structural alterations in breast stromal and epithelial DNA: the influence of 8,5'-cyclo-2'-deoxyadenosine
Published: 6/1/2006
Authors: K. M. Anderson, Pawel Jaruga, C. R. Ramsey, N. K. Gilman, V. M. Green, S. W. Rostad, J. T. Emerman, M Miral Dizdar, D. C. Malins
Abstract: (5'S)-8,5'-Cyclo-2'-deoxyadenosine (S-cdA), which arises from the reaction of the hydroxyl radical (*OH) with 2'-deoxyadenosine in DNA, is a lesion comprising a base-sugar linkage that distorts the DNA backbone. This structure impedes transcription a ...
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=907940

35. Repair of Formamidopyrimidines in DNA Involves Different Glycosylases - Role of the OGG1, NTH1, and NEIL1 Enzymes
Published: 12/9/2005
Authors: J Hu, N de Souza-Pinto, K Haraguchi, Barbara A Hogue, Pawel Jaruga, M M Greenberg, M Miral Dizdar, V. Bohr
Abstract: 2,6-diamino-4-hydroxy-5-formamidopyrimidine (FapyG) and 4,6-diamino-5-formamidopyrimidine (FapyA), are formed abundantly in DNA of cultured cells or tissues exposed to ionizing radiation or to other free radical-generating systems. We show here that ...
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=830498

36. Mouse NEIL1 protein is specific for excision of 2,6-diamino-4-hydroxy-5-formamidopyrimidine and 4,6-diamino-5-formamidopyrimidine from oxidatively damaged DNA
Published: 12/21/2004
Authors: Pawel Jaruga, M Birincioglu, T A Rosenquist, M Miral Dizdar
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=903209

37. Oxidative DNA Damage: Induction, Repair and Significance
Published: 9/1/2004
Authors: M D Evans, M Miral Dizdar, M S Cooke
Abstract: The generation of reactive oxygen species may be both beneficial to cells, performing a function in inter- and intra-cellular signaling, and detrimental, modifying cellular bimolecules, accumulation of which has been associated with numerous diseases ...
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=830412

38. Overexpression and Rapid Purification of Escherichia Coli Formamidopyrimidine-DNA Glycosylase
Published: 3/1/2004
Authors: Prasad T Reddy, Pawel Jaruga, T R O'Connor, H Rodriguez, M Miral Dizdar
Abstract: Formamidopyrimidine DNA glycosylase (Fpg) is a DNA glycosylase with an associated AP lyase activity. As a DNA repair enzyme, Fpg excises several modified bases from DNA associated with exposure to oxidizing agents such as free radicals. Experiments ...
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=830402

39. Substrate specificities and excision kinetics of DNA glycosylases involved in base-excision repair of oxidative DNA damage
Published: 10/29/2003
Author: M Miral Dizdar
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=903147

40. DNA Base Damage by the Antitumor Agent 3-Amino-1,2,4-Benzotriazine 1,4-Dioxide (Tirapazamine)
Published: 9/24/2003
Authors: M Birincioglu, Pawel Jaruga, G Chowdhury, H Rodriguez, M Miral Dizdar, K Gates
Abstract: Tirapazamine is a bioreductively activated DNA-damaging agent that selectively kills the hypoxic cells found in solid tomors. This compound shows great clinical promise and is currently being examined in a variety of clinical trails, including sever ...
http://www.nist.gov/manuscript-publication-search.cfm?pub_id=830363



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